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Iron and ferroptosis control For iron and ferroptosis control, the rationale is that the labile iron pool (LIP) catalyzes H 2 O 2 OH via Fenton/HaberWeiss chemistry and, when PUFA-containing phospholipids (PUFA-PLs), NOX/LOX activities and GPX4 insufficiency coincide, licenses ferroptosisa lipid peroxidationdriven cell death pathway (Jiang et al

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Furthermore, it prevented apoptosis and reduced the expression of microRNA (miR-339-5p) that is involved in programmed cell death

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Another contributing factor to cataract formation is ascorbic acid, a powerful antioxidant that suffers from the same age-related decline as GSH [10]

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