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Y., Wada, S., Sato, K., & Higashi, A

concurrently, BNIP3 recruits Parkin to mitochondria to initiate autophagic flux, whereas NIXas a ubiquitination substrate of Parkinundergoes conformational remodeling post-modification to specifically engage adaptor protein NBR1, ultimately enabling targeted autophagic degradation of compromised mitochondria (Wang et al., 2023)

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Enhanced monoacylglycerol lipolysis by ABHD6 promotes NSCLC pathogenesis

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In fact, more than half of all acute liver failure cases in the US are due to acetaminophen overdose