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They could decrease the expression and phosphorylation of epidermal growth factor receptor (EGFR), Met, Akt, and NF-B in cervical cancer cells, and totally inhibit the EGFR/Met-Akt/NF-B signaling pathway activated by hepatocyte growth factor (HGF) and epidermal growth factor (EGF) (69)

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doi: 10.1001/jama.264.1.48 146 LevinePHJacobsonSPocinkiAGCheneyPPetersonDConnellyRRet al

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