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Another consequence of a compensatory increased in local IGF-I activity is the finding of normal childhood and pubertal linear growth despite endocrine IGF-I deficiency in type 1 diabetes (86)

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[11] Brand-Miller et al., (2002) Glycemic index and obesity, The American Journal of Clinical Nutrition, Volume 76, Issue 1, Pages 281S-285S, ISSN 0002-9165, ajcn/76.1.281S

American Family Physician, 72(7), pp.12851292.

Their effects on the individual components of MACE, myocardial infarction (reduced in LEADER and Harmony Outcomes), and stroke (reduced in SUSTAIN-6 and REWIND), varied in each trial (Table 1)