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This paper intends to address the pathogenesis of oxidative stress injury and schizophrenia, and the significance of oxidative stress in the treatment of schizophrenia

We quantified the trade-offs between efficiency and cost, considering RGD-GHK for the following experiments

DRP1 upregulation promotes pancreatic cancer growth and metastasis through increased aerobic glycolysis

Senescent stimuli endow cells with resistance to apoptosis, which involves high activity of anti-apoptotic molecules, such as BCL2 and BCL-XL, and reduced pro-apoptotic BAX protein