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By knocking out the Uox gene, these mice completely lose the ability to degrade uric acid, resulting in a significant spike in serum uric acid levels and the spontaneous formation of hyperuricemia and urate nephropathy phenotypes [6-7]

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164 The carnitine palmitoyl transferases (CPT), the rate-limiting enzymes in the FAO pathway, transport long-chain acyl-CoA from the cytosol to mitochondria

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