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Neuroscientific research has further examined the peptides interaction with monoaminergic pathways, particularly those involving dopamine and serotonin signaling

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NIX can directly bind to LC3 in the BH3 domain, recruiting phagosomes (precursors of autophagosomes) to mitochondria and acting as autophagy receptors, mediating mitochondrial clearance after mitochondrial damage and red blood cell differentiation [71]

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