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Overproduction of reactive oxygen species (ROS) in the nucleus pulposus cells (NPCs) is a critical contributor to the progression of IDD through increasing oxidative stress in the microenvironment of the extracellular matrix (ECM) 5 , leading to ferroptosis and long-term persistent inflammation 6,7 , which would cause the loss of structure and function in the intervertebral disc (IVD) ultimately

Conversely, avoiding excessive or improper loading that could disrupt these pathways is crucial

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Have you taken before photos to track your progress over months, not days