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These observations become important later when deciding whether to extend any escalation step

Binding studies in CHO-K1 cells expressing GLP-1R showed that lixisenatide is a potent and selective GLP-1RA, with a binding affinity to GLP-1R approximately four times greater than that of GLP-1 (88)

7 In patients with T2DM, the release of incretin hormones is physiologically insufficient, thereby compromising the ability to adequately secrete insulin and also execute other physiologic processes required to meet the metabolic requirements to maintain normoglycemia

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Its mainly the C10 fat (capric acid) that may boost GLP-1