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Despite a limited amount of data, it is reasonable to postulate that dysregulated control of macrophage survival in an inflamed environment could exaggerate inflammation, and this could be relevant to the initiation or progression of CD

doi: 10.1007/5584_2020_496

Fisetin, an inhibitor of cyclin-dependent kinase 6, down-regulates nuclear factor-kappaB-regulated cell proliferation, antiapoptotic and metastatic gene products through the suppression of TAK-1 and receptor-interacting protein-regulated IkappaBalpha kinase activation

More broadly, they challenge the traditional separation between metabolic and psychiatric disease, suggesting peripheral metabolic signals can influence central processes such as motivation and behavior

2017;68:22432.e224

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