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This hormone, in turn, inhibits osteoclastic activity, which decreases the release of calcium from the bone into the bloodstream, leading to decreased bone resorption [56, 57]

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Other interesting contributors are the complement proteins C1q and CR3 (Veerhuis et al., 2011) which are highly produced in the microglia as crucial factors in synapse pruning and are highly present in CSF of AD patients with emerging evidence about their role in A pathogenesis (Fatoba et al., 2021)