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Furthermore, enhanced glutamine metabolism has been shown to significantly increase -KG production, which, in turn, activates the mTOR1 signaling pathway and further inhibits the autophagy-promoting kinase (APCK) of the ULK1 complex by mediating site-specific phosphorylation of ULK1 (Ser637 and Ser757) and Atg13 (Ser258) activity, thereby promoting cancer cell proliferation [27]

B Cell Therapies: Rituximab (Rituxan), Ocrelizumab (Ocrevus) Rituximab and ocrelizumab, anti-CD20 B cell depleting monoclonal antibodies, have become critical agents in the management of MS, underlying the importance of B cells in the pathophysiology of MS

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Effect of flavonoids rich extract of Capparis spinosa on inflammatory involved genes in amyloid-beta peptide injected rat model of Alzheimer's disease