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The cascade of events causing liver injury begins with the physiological dietary intake and intestinal absorption of copper, which cannot be adequately managed by patients with Wilson disease, ultimately resulting in severe copper dysregulation
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69 Potential mechanisms include decreased intestinal lipid production, reduced intestinal triglyceride absorption due to lowered secretion of apoB48-containing chylomicrons, smaller postprandial triglyceride secretion and effects on the expression of genes involved in lipid metabolism
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