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Of note, the changes registered through the follow-up were comparable between men and women

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however, genetics [2] and hormonal mechanisms [3] have been implicated

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The findings demonstrate that the neuromodulatory molecule, glucagon-like peptide-1 (GLP-1), secreted by colonic enteroendocrine L-cells in response to the bacterial metabolite, indole, stimulated colonic vagal afferent activity