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Day JW, Ottaway N, Patterson JT, Gelfanov V, Smiley D, Gidda J, et al
Since an IGF-1R construct, in which the tyrosine at position 1136 was replaced by a phenylalanine, also led to a strong inhibition of phosphorylated Akt in transfected cells, it was suggested that this mechanism may be responsible for the apoptotic effect of PPP (Vasilcanu et al, 2004)
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Mitochondrial dysfunction, which leads to increased ROS formation and thus cell apoptosis and impaired energy metabolism glucose, fatty acid, and glutamine metabolism contribute to OA pathogenesis (201203)
A wide spectrum of histologic patterns of senescence may be seen in benign melanocytic lesions
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