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Estrogen receptor activation of phosphoinositide-3 kinase, akt, and nitric oxide signaling in cerebral blood vessels: rapid and long-term effects

Our transcriptomic and functional analyses further revealed the molecular basis of this crosstalk

CD95-resistant and Bcl-x L overexpressing CEM cells exhibited higher intracellular GSH levels in comparison to parental cells

overexpressed miR-340-5p in the myocardium by direct myocardial injection of AVV-9 containing miR-340-5p precursor, which targeted inhibition of MyD88 and thus restored declining GSH levels, alleviated insult of cardiac function and oxidative injury induced by sepsis in the hearts of mice [253]