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In AD model mice overexpressing human mutant APP, the knockout of endogenous ApoE leads to a significant reduction in A deposition in the brain, along with lowered A40 and A42 deposits, underscoring the substantial role of ApoE in A fibril formation and amyloid deposition (Holtzman et al., 2000

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Clin Neurol Neurosurg 2005

Type 2 Diabetes and Metabolic Syndrome Studies : Examining changes in HbA1c, fasting glucose, insulin-related measures, and other metabolic readouts in research populations with excess weight

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