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A striking illustration of gutbrain neural connectivity is seen in PD: misfolded -synuclein protein aggregates, a hallmark of PD, are hypothesized to originate in the gut and spread to the brain via vagal nerve fibers in a prion-like fashion [31]

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Activation of this receptor also slows gastric emptying, promotes satiety, and reduces food intake, contributing to weight management and appetite control

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Second, it is considerably more aggressive than its skin counterpart , with a higher rate of spread to lymph nodes and distant organs at the time of diagnosis