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Under hypoxic conditions, the reduced oxygen availability diminishes the activity of prolyl hydroxylase domain enzymes (PHDs), which typically employ oxygen and -ketoglutarate as substrates to hydroxylate HIF-1, thereby targeting it for degradation

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Serra et al., 2009), and vascular dementia (Famulari et al., 1996, Dominguez et al., 2008

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This is why a low and slow approach to dosing is critical