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LL37 and released DNA can form a complex, which can activate plasmacytoid dendritic cells via internal Toll-like receptors (TLR), which then instruct T-lymphocytes to become Th1 and Th17 cells and induce production of several cytokines

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This reversal is indicative of severe metabolic stress and highlights the cells prioritization of maintaining membrane potential over efficient ATP production (Klukov et al