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Recently, there is a theory explaining how NRF2 is stabilized under stress conditions, called the two-site substrate hinge and latch model. It is known that each of the two dimeric KEAP1 molecules will bind NRF2 separately through their respective Kelch repeat domain to either the ETGE or DLG motifs in NRF2

Beumer J, Artegiani B, Post Y, Reimann F, Gribble F, Nguyen TN, et al

In addition to the effect on food intake reduction and glucose-stimulated insulin secretion, GLP-1 also acts to delay gastric emptying therefore aiding satiety and glycaemic control (42)

It was a big part of the expression of the theme in Europe last year, but I think it will gain relevance in the U.S

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