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In summary, GSH emerges as a central suppressor of cuproptosis by buffering cytosolic and mitochondrial copper and by integrating with metallothioneins and stress-responsive transcriptional programs

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doi: 10.1016/j.anai.2023.10.032 154 PazukhinaEAndreevaMSpiridonovaEBobkovaPShikhalevaAEl-TaraviYet al

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Moreover, it was shown that the lack of PLP in the diet resulted in reduced fecal H 2 S level by approximately 50% [39]