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Immunology 53:29197.[PubMed], [Web of Science ] , [Google Scholar]) postulate that this biphasic response is due to low doses of CGN stimulating a population of suppressor macrophages that actively suppress T lymphocyte proliferation, while higher doses abolish suppressor activity via cytotoxicity

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This study provides insight into how differential dynamics of peptideligand engagement with the GLP-1R TMD can promote differences in G protein-mediated signalling, improving molecular understanding of mechanisms that contribute to ligand-dependent differential efficacy at the GLP-1R

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