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In contrast, CCAS is primarily and directly defined as a neuropsychiatric manifestation of cerebellar damages that include significant cerebellar neuropathologies, although many individual components of CCAS are present among signs of other psychiatric syndromes [6, 9, 12]

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63 Moreover, mucosal inflammation in claudin-7 KO mice appears to be a product of epithelial cell instability and not necessarily the result of barrier deregulation, given that no apparent gut leakiness was documented at early time points in these mice

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The mouse model used for these experiments is a general knockout and we can therefore discount the possibility that GLP-1 exerts its inhibitory action by paracrine effects secondary to activation of GLP-1Rs in delta and beta cells, suggesting that GLP-1s glucagonostatic effect involves a receptor distinct from GLP-1R