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While this method addresses the issue of copper deficiency, further research is required to determine if it can also address excessive copper ion levels

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If the chronic cerebral hypoperfusion hypothesis is correct then brain neuronal accumulations of A1-42 and tau proteins must be considered downstream consequences rather than causes of AD [48]

The cost is additive, not the risk

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2 and Table 3 provide a summary of the known/proposed G-protein coupled receptors mediating the effects of the select neuropeptides