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Patients on GLP-1 medications may be losing muscle along with fat, potentially leading to frailty despite a lower weight

Evidence level is moderate-high with more advanced Phase 3 enrollment than retatrutide

Limited or virtual only programs with no physician follow up

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The potential molecular mechanisms underlying these effects included disturbances in redox homeostasis (as evidence by the enhanced production of ROS and reactive nitrogen species), failure of enzymatic and nonenzymatic cytoprotective mechanisms involving the glutathione system as well as nuclear factor kappa B (NF-B) and fibroblast growth factor 2-mediated pathways, and impaired mitochondrial function