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We adopted a male-only design in this preliminary mechanistic investigation to reduce endocrine-related variability associated with the estrous cycle and estrogen, which can modulate microglial activation and neuroinflammatory responses, thereby facilitating more stable identification of gutbrain inflammatory signaling and glial lipotoxic mechanisms following PBAT-MPs exposure

Each has its own trade-offs

The injection site and method are determined by your provider at intake

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