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Retrieved from Sahi, N., Nguyen, R., Patel, P., & Santos, C

Circuit analysis revealed the medial NAc shell VTA population to be modulated by exendin-4(Wang et al., 2015)

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Emerging findings suggest the metabolic switch from glucose to fatty acid-derived ketones represents an evolutionarily conserved trigger point that shifts metabolism from lipid/cholesterol synthesis and fat storage to mobilization of fat through fatty acid oxidation and fatty-acid derived ketones, which serve to preserve muscle mass and function

Ubiquitination also plays a central role in controlling YAP1 levels