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Here, we demonstrate that TMAO activates aortic VICs towards a myofibroblastic phenotype through the induction of endoplasmic reticulum stress

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Investigations purport that the peptide blend may be used in immune cell cultures (e.g., lymphocytes, monocytes) or organoid models to examine how enhanced GH axis signaling modulates cytokine production, cell proliferation, or oxidative stress under inflammatory stimulus

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