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IGF1R inhibitors inhibits radiation-induced DNA damage repair by prolonging the expression of phosphorylated histone H2AX, and interfering with the Ku-DNA binding and Ku70/Ku80 expressions (Katayama et al., 2007)

Classic explanation for such deviations exist: LQ model fails beyond about 3 decades of cell killing models quadratic component causes it to over-predict the lethality of large radiation doses

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