In PACs stimulated with cerulein and resistin, pro-inflammatory processes are activated, but -lipoic acid, via PPAR- activation, increases HO-1 and catalase expression, suppresses ROS and IL-6 levels, and thereby indicates functional integration of peroxisomal and mitochondrial antioxidant defenses ( This limitation is particularly relevant when extrapolating these findings to HTG-AP, because hyperlipidemic disease states impose continuous fatty acid excess and may shift the mitochondriaperoxisome relationship from adaptive cooperation to maladaptive overload
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Metabolomic similarities between oral candidiasis and xerostomia were evident, with shared markers such as L-valine, L-leucine, D-proline and 4-hydroxyphenylpyruvic acid
Consumption of a high fat diet results in a surplus of NADH and FADH 2 that then increases the flux through the ETC with a resultant increase in ROS generation
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