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A study on genetic linkages between aberrant lipid metabolism and PD directly demonstrates that glucocerebrosidase and sphingomyelin phosphodiesterase 1 mutations enhance PD risk in humans, and that galactosylceramide beta hydrolase function loss increases -synuclein aggregation and accumulation in experimental murine models

As the medical community's understanding of what drugs are used for COVID long haulers rapidly evolves, NAC is frequently being incorporated into cutting-edge, multi-disciplinary treatment protocols