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GSH supplementation prevented their induction in response to cystine starvation, but this effect was abrogated by OU749 (Fig

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Therapeutic Advances in Endocrinology and Metabolism, 15, 2042018823122236720420188231222367

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Primary Mechanisms Investigated in Research G-Actin Sequestration: TB-500s primary mechanism is binding to monomeric G-actin via its LKKTET motif, preventing polymerisation

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