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Protection from toxic proteins: These drugs may reduce accumulation of beta-amyloid and tau proteins in the brain

The end-result of SCD1 inhibition may, therefore, be an aggravation of NASH due to intracellular accumulation of cytotoxic saturated fatty acids [51, 62], placing the partitioning of saturated fatty acids to mono-unsaturated fatty acids as a protective factor in delaying NAFLD progression

Glucagon agonism raises theoretical concerns about hyperglycemia in insulin-resistant populations, though fasting glucose remained stable or improved across all retatrutide arms

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PMID: 31 KlingbergEBilbergABjrkmanSHedbergMJacobssonLForsblad-dEliaHet al