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Inhibition of kynurenine 3-monooxygenase (KMO) significantly suppressed XA elevation and attenuated both oxygen-glucose deprivation (OGD)-induced cardiomyocyte injury and ligation-induced myocardial ischemia injury, suggesting XA's potential utility as a ferroptosis marker in CAD
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doi: 10.1038/s41401-020-00589-x 49
Participants received pharmaceutical-grade product with verified purity
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consistency and progressive overload matter more than session length
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