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These include enhancing intracellular TH availability via modulation of transporters such as monocarboxylate transporter 8 (MCT8) and deiodinases (e.g., DIO1), as well as directly targeting ECM remodeling through upregulation of MMP activity, inhibition of HA synthesis, and blockade of integrin-mediated signaling pathways (119121)
Fras JP, Davies MJ, Rosenstock J, et al
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[DOI] [PMC free article] [PubMed] [Google Scholar] 85.Ufnal M, Pham K
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