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Therefore, the reduction of its activity can cause insufficient ATP production

Although further research is required, it is possible that acetylation of mutant p53 by aspirin, in some cases, may restore its DNA-binding properties leading to target gene expression

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3.2 Gut microbiota alterations in OA 3.2.1 Gut microbial dysbiosis in OA Recent years have seen many novel studies unveiling the important role of gut microbiota dysbiosis in OA pathogenesis, with strong evidence for the gut-joint axis hypothesis of OA