During development and progression of atherosclerosis, Nrf2 signaling modulates many physiological and pathophysiological processes, like regulation of lipid homeostasis, CD36 gene expression regulation, foam cell formation, macrophage polarization, immunity regulation (Th2 differentiation and inhibition of pro-inflammatory gene expression through NFB down-regulation), redox regulation and inflammation, improvement of endothelial dysfunction, as well as GSH synthesis and utilization (234245)
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For example, guanosine is the most frequent nucleobase at a specific base position in the genome, but there is a SNP in which the guanosine is replaced by adenine
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This effect was followed by the increase in substrate fluorescent or GSH transport and decreasing of GSSG efflux
