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Thus, we speculate that T3 can increase the production of non-12-OH FXR-antagonistic BAs in human likely attributed to the same mechanisms in mice, thereby promoting GLP-1 and insulin production by suppressing intestinal FXR signaling

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These observations suggest that the increase in tau phosphorylation induced by palmitoyl-L-carnitine is mediated by a network of tau kinase pathways associated with calcium overload

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