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As a substrate for glutathione peroxidase, GSH participates in the reduction of hydrogen peroxide and lipid hydroperoxides, protecting membranes from oxidative damage

High-fat diet (HFD)-induced diabetic mice exhibited more severe renal damage, including higher renal injury scores, elevated levels of serum BUN (blood urea nitrogen), CCr (creatinine clearance rate) and Cysc (cystatin C), oxidative stress and ferroptosis