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Activation of the PP2A-B56 heterocomplex synergizes with venetoclax therapies in AML through BCL2 and MCL1 modulation

Dual depletion of NHE1 and SWELL1 markedly suppresses breast cancer cell migration, extravasation, and metastasis, underscoring the pivotal role of this migration mechanism in breast cancer progression [163]

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140 Whereas the continuous supply of FeS clusters by NFS1 also prevented the iron-starvation response from being triggered in lung adenocarcinomas, NFS1 suppression was shown to predispose cancer cells to ferroptosis