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GSH behaves as redox sensor at the DNA synthesis onset by maintaining nuclear architecture providing the appropriate redox environment for DNA replication and safeguarding DNA integrity (72) and is a key regulator of epigenetic events critical in cell proliferation regulation and cellular resistance to apoptosis (73)

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Endogenous DHHC9 KO substantially blocked GLUT1 palmitoylation (Fig

Treatments were performed by a single researcher and continued until mice were 420-days-old

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The only catch is that HA isn't a single thing