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Ismail Kimji, MD, FRCPC

CNS injury-induced astrocyte activation has strengthened the view that many neurodegenerative diseases involve non-cell-autonomous mechanisms, where glial cells, especially astrocytes, actively shape disease progression

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This process, along with GCL inhibition and the formation of mixed disulfides between GSH and protein sulfhydryl, concurred to a steady-state decrement of free GSH concentration and, consequently, to an intracellular redox state shift toward more oxidizing conditions, which could be required for avoiding fast reduction of oxidized cysteines allowing the upstream ROS-induced autophagic stimulus to be propagated

More recent research has pushed into less expected territory