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This intestinal permeability allows partially digested food particles, bacterial endotoxins, and other inflammatory triggers to cross the intestinal barrier and enter the bloodstream

When exposed to a hyperglycemic milieu, the GIPR was found to downregulate in a study of pancreatic islet cells (59) and the patients with T2DM might express a small amount of GIPR or defective GIPR (60, 61)

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[DOI] [PubMed] [Google Scholar] 48.Williamson D., Hargreaves R