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The present study provides evidence supporting the idea that hyperinsulinemic condition causes neuronal insulin resistance, as indicated by the downregulation of insulin signalling molecules, including PI3K/AKT, with the upregulation of GSK-3 and TTBK1
[DOI] [PubMed] [Google Scholar] 50.Goggin JM, Biller DS, Debey BM
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View in: Pubmed Blockade of TIM-1 on the donor graft ameliorates graft-versus-host disease following hematopoietic cell transplantation
Mitochondrial dysfunction in airway disease
Many of these receptors are clathrin-dependent endocytosed, one example being LRP1 which is responsible for the endocytosis of amyloid-beta and APOE 2 and 3 [12]
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