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Upon release, it binds selectively to GLP-1 receptors, which are widely distributed throughout the body, including the GI tract, pancreas, brainstem, hypothalamus, and vagal afferent nerves [27,28,29,30]
Regulation of small GTPases by GEFs, GAPs, and GDIs
The observed interactions between the ECD and ECL1/3 in the inactive GLP-1R structure presented here agree with previous studies suggesting that the ECD stabilized an inactive state of the glucagon receptor through interactions with ECL1 and ECL3 20
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