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Whereas, GA (Group IV) pretreatment significantly prevented ISO-induced overexpression of TNF-, IL-6, NF-B, COX-2 and iNOS in rat cardiac tissue (Figure 5A,B)

Inflammation cytokines (e.g., TNF-, IL-1) abundant during HIV infection exacerbate mitochondrial dysfunction and ROS production, forming a self-amplifying loop that increases mtDNA damage (85)