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In fact, B cell-deficient mice produce 1001000 times more infectious progeny in tissues during recurrent mouse CMV (MCMV) infection compared to control mice (Jonji et al, 1994), and adoptive transfer of memory B cells (MBCs) into Rag / mice is effective to provide long-term protection against an ongoing lethal MCMV infection (Klenovsek et al, 2007)

These results suggest that GpoA is critical not only for resistance to oxidative stress but also for effective invasion and systemic dissemination during infection

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While full GLP-1R agonism drives the majority of GI side effects (nausea, vomiting), the partial agonism at GLP-1R combined with GIPR-mediated complementary effects may achieve greater overall efficacy with proportionally less GI distress [7]