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New pathways for reactive oxygen species generation in inflammation and potential novel pharmacological targets

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Nandi et al., 2020)

Under 18 years of age

doi: 10.1111/bpa.12228 55 ItalianoAMichalakSSouliPPeyronACPedeutourF

In this paper, we explore the potential role of certain amyloidogenic proteins, including amylin, amyloid beta, the prion protein, huntingtin, and -synuclein, in disease processes that result in the accumulation of deposits of protein fibrils, along with lipid membrane components of damaged mitochondria, which we argue may be a mechanism to sequester deuterium in order to reduce the deuterium burden in the tissues