Yesbut with reasonable expectations
Oxidative stress-induced mitochondrial DNA damage in human retinal pigment epithelial cells: a possible mechanism for RPE aging and age-related macular degeneration
Studies conducted in mice and human foreskin fibroblasts have revealed that S-acetyl-glutathione molecules are taken up directly by cells with subsequent conversion to glutathione by cleavage of the acetyl bond within the cell
49(11): p
Additionally, BPC-157 modulates the adrenaline and noradrenaline systems by counteracting both beta-adrenergic overstimulation (e.g., isoprenaline) and blockade (e.g., sotalol), preventing resultant hemodynamic collapse, multiorgan failure, and vascular occlusion-like syndromes in rodent models [41, 48, 49]
These studies on myocardial injury and the GSH system reveal new roles of these clinical drugs in the treatment and further elaborate their mechanisms, which provide new ideas for their clinical application